We don’t know how this would pan out in humans, but in mice it sounds nice: Researchers found that mice lacking a certain pain receptor live longer.
Study authors genetically engineered mice so they wouldn’t have TRPV1 pain receptors.
Normally, such receptors get activated as a result of high temperatures or hot chili peppers, New Scientist reported.
Male mice without these receptors lived 12% longer than those that had them.
The outcome for female mice was even more promising: Those that lacked the receptors lived 16% longer.
It appears that those without the receptor also produced more insulin.
On the flip side, mammals have these pain receptors so they will be biologically warned about dangerous objects and situations.
“Pain is very important for animals living in the wild and probably outweighs the benefits of a youthful metabolism,” Andrew Dillin at the University of California, Berkeley, told New Scientist.
Still, there could be therapeutic applications that stem from these insights.
To read more on this study, click here.